The Benefits of MCT Oil: Successful Aging!

The Hard Truth.

Cancer:

  • 1 out of every 3 people in the U.S. will eventually develop some form of cancer in his or her lifetime
    • Women: 1 in 8 chance of developing breast cancer
    • Men: 1 in 9 chance of developing prostate cancer

Diabetes:

  • Total Prevalence of Diabetes: 25.8 million children and adults in the U.S. – 8.3% of the population
    • Prediabetes and at Risk of Developing Type II Diabetes: 79 million people
    • New Cases: 1.9 million people per year

Fat Chance.

Healthy Immune System. A healthy immune system decreases the risk of developing cancer. Yet, when germ exposure becomes excessive, causing great stress on the immune system, white blood cells become less efficient at eliminating cancer cells is reduced.

Medium-chain fatty acids (MCFAs) that come from MCT oil and coconut oil relieve stress on the immune system by extinguishing many invading microbes. This allows more white blood cells to seek out and destroy the cancerous cells in our bodies.

KetosisAll body cells, including cancer cells, are fueled by glucose. However, cancer cells have one fatal flaw – they do not have the metabolic capability to utilize fat as fuel like other cells.

Entering a state of ketosis with MCT Lean MCT Oil enables the body to start using fat for fuel rather than carbohydrates. When fats become the body’s primary source of energy, cancer cells may be starved out, as they are no longer supplied their necessary fuel to grow.

Can’t Stop The Power. 

Diabetes arises when the cells’ ability to respond to insulin and uptake glucose for fuel is severely diminished. MCFAs counteract this by supplying much needed energy to our cells regardless of insulin. Combined with a nutrient dense diet, MCFAs regulate blood sugar and speed up metabolism, helping maintain normal levels.

***Please consult your physician before use.***

References:

Berry, E. M. (1997). Dietary fatty acids in the management of diabetes mellitus. American Journal of Clinical Nutrition, 66.

Cohen, L. A. (1988). Medium chain triglycerides lack tumor-promoting effects in the n-methylnitrosourea-induced mammary tumor model. In The pharmacological effects of lipids, vol. 3, edited by J. J. Kabara. Champaign, Illinois: The American Oil Chemists’ Society.

Cohen, L. A. & Thompson, D. O. (1987). The influence of dietary medium chain triglycerides on rat mammary tumor development. Lipidsm 22(6).

Fine, E. J., Miller, A., Quadros, E. V., Sequeria, J. M. & Feinman, R. D. (2009). Acetoacetate reduces growth and ATP concentration in cancer cell lines which over-express uncoupling protein 2. Cancer Cell International, 9(14).

Fine, E. J., Segal-Isaacson, C. J., Feinman, R. D…Sparano, J. A. (2012). Targeting insulin inhibition as a metabolic therapy in advanced cancer: a pilot safety and feasibility dietary trial in 10 patients. Nutrition, 10.

Mercola, J. (2013, March). Ketogenic Diet May Be Key to Cancer Recovery. Mercola.com. Retrieved from http://articles.mercola.com/sites/articles/archive/2013/03/10/ketogenic-diet.aspx

Reddy, B. S. (1992). Dietary fat and colon cancer: Animal model studies. Lipids, 27(10).

Seyfried, T. N., Mukherjee, P., Kalamian, M. & Zuccoli, G. (2011). Restricted Ketogenic Diet (RKD) as an Alternative Strategy for GBM. Treatment Strategies, Oncology, 2(1).

Seyfried, T. N., Kiebish, M., Mukherjee, P. & Marsh, J. (2008). Targeting energy metabolism in brain cancer with calorically restricted ketogenic diets. Epilepsia, 49(8).

*These statements have not been evaluated by the Food and Drug Administration. These products are not intended to diagnose, treat, cure, or prevent any disease. 

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