The Benefits of MCT Oil: Brain Power!

The Fundamental Problem. Studies indicate that neurodegenerative disorders cause the brain to ineffectively utilize blood glucose as an energy source. This defect in energy conversion starves the brain of nourishment and weakens the ability of brain cells to withstand normal oxidative stress from free radicals. Consequently, the brain begins to age rapidly and eventually degenerate into full dementia.

An Alternative Fuel Source. Ketones are designed to fuel the brain and other peripheral organs during a reduced carbohydrate diet when blood glucose levels are low. Normally, our brains derive all of its energy requirements from glucose, but when ketosis is induced, up to 75% of energy requirements can be obtained through the production of ketones. Medium chain triglycerides (MCTs) lead to substantially more ketones in the blood compared to long chain triglycerides (LCTs), making it a critical substance for a healthy brain.

Defense Against Brain Disease. Studies show a state of ketosis may help prevent and provide symptomatic relief to a wide variety of brain disorders:

  • Parkinson’s Disease
  • ALS
  • Huntington’s Disease
  • Traumatic Brain Injury
  • Autism
  • Alzheimer’s Disease

Ketones have been found to reduce Alzheimer’s-type plaque in the brain. In clinical studies, Alzheimer’s patients who consumed a beverage with MCTs compared to one without MCTs, scored significantly better on cognitive tests. In animal studies, ketosis has led to improved performance on visual-spatial memory tasks, increased ability of learning tasks, and an enhanced short-term memory.

Providing Some Relief. Ketones trigger the activation of specialized proteins called brain-derived neurotrophic factors (BDNFs) that aid in brain cell maintenance, repair, and protection. BDNFs also stimulate the growth of new brain cells to replace dead or dying cells, allowing some mental function restoration.

References:

Constantini, L. C., Barr, L. J., Vogel, J. L. & Hendersen, S. T. (2008). Hypometabolism as a therapeutic target in Alzheimer’s disease. BMC Neuroscience, 9.

Duan, W., Guo, Z., Ware, M., Li, X. J. & Mattson, M. P. (2003). Dietary restriction normalizes glucose metabolism and BDNF levels, slows disease progression, and increases survival in huntingtin mutant mice. Proceedings of National Academy of Sciences USA, 100(5).

Fife, B. (2004). The Coconut Oil Miracle. New York: Avery.

Fife, B. (2012). Coconut Ketones: Fueling Brain Function & Reversing Autism. Well Being Journal, 21(5).

Fife, B. (2012). Conquering Alzheimer’s with Coconut Ketones. Coconut Research Center.

Gasior, M., Rogawski, M. A. & Hartman, A. L. (2006). Neuroprotective and disease-modifying effects of the ketogenic diet. Behavioural Pharmacology, 17.

Reger, M. A., Hendersen, S. T., Hale, C., Cholerton, B. Baker, L. D., Watson, G. S., Hyde, K., Chapman, D. & Craft, S. (2004). Effects of beta-hydroxybutyrate on cognition in memory impaired adults. Neurobiology of Aging, 25(3).

Tieu, K., Perier, C., Caspersen, C., Teismann, P., Wu, D. C., Yan, S. D., Naini, A., Vila, M., Jackson-Lewis, V., Ramasamy, R. & Prezborski, S. (2003).D-beta-hydroxybutyrate rescues mitochondrial respiration and mitigates features of Parkinson disease. Journal of Clinical Investigation, 112(6).

Van der Auwera, I., Wera, S. Van Leuven, F. & Hendersen, S. T. (2005).A ketogenic diet reduces amyloid beta 40 and 42 in mouse model of Alzheimer’s disease. Nutrition & Metabolism (London), 2.

Zhao, Z., Lange, D. J., Voustianiouk, A., MacGrogan D, Lo, H., Suh, J. Humala, N., Thiyagarjan, M., Wang, J. & Pasinetti, G. M. (2006). A ketogenic diet as a potential novel therapeutic intervention in amyotrophic lateral sclerosis. BMC Neuroscience, 7.Zhao, Z., Varghese, M., Vempati, P., Dzhun, A., Cheng, A., Wang, J., Lange, D., Bilski, A., Faravelli, I. & Pasinetti, G.M. (2012).Caprylic triglyceride as a novel therapeutic approach to effectively improve the performance and attenuate the symptoms due to the motor neuron loss in ALS disease. PLoS One, 7(11).
*These statements have not been evaluated by the Food and Drug Administration. These products are not intended to diagnose, treat, cure, or prevent any disease.

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